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Integrative multi-omics identifies AP-1 transcription factor as a targetable mediator of acquired osimertinib resistance in non-small cell lung cancer

GSE295823 Homo sapiens Genome binding/occupancy profiling by high throughput sequencing; Expression profiling by high throughput sequencing 20 samples Submitted 2025/05/27 Platform GPL24676Platform GPL34281
SuperSeries — this record groups several sub-series.
Summary
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Published in
Integrative multi-omics identifies AP-1 transcription factor as a targetable mediator of acquired osimertinib resistance in non-small cell lung cancer
Dayanc B, Eris S, Gulfirat NE et al. · Cell death & disease 2025 · PMID 40414926 · doi:10.1038/s41419-025-07711-z
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Direct links to NCBI, no account and no request form: the whole study as GSE295823_RAW.tar, processed values as the series matrix, the supplementary file directory, and per-sample supplementary files for any of the 20 samples. Raw sequencing reads are also available from ENA.

Also filed as BioProject PRJNA1256437 and SRA study SRP535260. Searching any of these in the dataset finder brings you back here.

Study design
7 conditions, mostly in duplicate
HCC827 cells, control, ×5 Resistant HCC827 cells, (HCC827-OsiR), ×5 Resistant HCC827 FOSL1 g1 knockout cells, (HC… ×2 Resistant HCC827 FOSL1 g2 knockout cells, (HC… ×2 Resistant HCC827 JUN g1 knockout cells, (HCC8… ×2 Resistant HCC827 JUN g2 knockout cells, (HCC8… ×2 Resistant HCC827 Renilla knockout cells, (HCC… ×2

Supports a case/control comparison: 10 samples read as cases, 5 as controls.

7 replicated groups read from 20 sample titles; they account for 20 of them. Check it against the sample list below before relying on it.

Samples in this study
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