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Targeting VRK2 Enhances the Efficacy of Anti-PD-1 Therapy in Hepatocellular Carcinoma by Reducing Direct Phosphorylation and Stability of MYC: CUT-TAG sequencing for MYC in hepatocellular carcinoma cells

GSE278073 Homo sapiens Genome binding/occupancy profiling by high throughput sequencing 16 samples Submitted 2025/05/22 Platform GPL24676
Summary
VRK2–MYC axis, a transcription-dependent regulatory axis, could be a prognostic indicator and potential therapeutic target in liver cancer.
Published in
VRK2 targeting potentiates anti-PD-1 immunotherapy in hepatocellular carcinoma through MYC destabilization
Su C, Liao Z, Mo J et al. · Nature communications 2025 · PMID 41073389 · doi:10.1038/s41467-025-64079-6
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Direct links to NCBI, no account and no request form: the whole study as GSE278073_RAW.tar, processed values as the series matrix, the supplementary file directory, and per-sample supplementary files for any of the 16 samples. Raw sequencing reads are also available from ENA.

Also filed as BioProject PRJNA1165406 and SRA study SRP534884. Searching any of these in the dataset finder brings you back here.

Study design
16 conditions, each sampled once — no replicated groups

Read from 16 sample titles: 16 distinct titles with little repetition. Check it against the sample list below before relying on it.

Samples in this study
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